If serotonin is bad:
1. Why do selective serotonin reuptake inhibitors treat premature ejucalation effectively ? This is a very objective measurement. Is premature ejaculation a sign of good health then ?
2. And glucose is good. Why then does glucose water raise serotonin in rats:
3. [A major decrease in 5-hydroxyindoleacetic acid was found in cortex and hippocampus in rats exposed to cadmium during lactation]http://www.sciencedirect.com/science/article/pii/S0892036296002188 So maybe cadmium has positive effects ?
4. [Serotonin in mammals is made by two different tryptophan hydroxylases: TPH1 produces serotonin in the pineal gland and the enterochromaffin cells, while TPH2 produces it in the raphe nuclei and in the myenteric plexus. Genetically altered mice lacking TPH1 develop progressive loss of heart strength early on. They have pale skin and breathing difficulties, are easily tired, and eventually die of heart failure.][http://www.ncbi.nlm.nih.gov/pmc/articles/PMC263847/]
I actually wanted to make a whole list about topics like
cold exposure/omega-3/melatonin/cortisol/cooked protein/amphetamines/exercise/acidbase theory/sleep duration/gut flora/nuts etc. all things where I just have questions.
Studies that I read in the last 5 years, or experiences that I have, that just don't fit in the ray peat theory. But reading those studies again takes quite some time... Maybe another day.
1. Why do selective serotonin reuptake inhibitors treat premature ejucalation effectively ? This is a very objective measurement. Is premature ejaculation a sign of good health then ?
2. And glucose is good. Why then does glucose water raise serotonin in rats:
3. [A major decrease in 5-hydroxyindoleacetic acid was found in cortex and hippocampus in rats exposed to cadmium during lactation]http://www.sciencedirect.com/science/article/pii/S0892036296002188 So maybe cadmium has positive effects ?
4. [Serotonin in mammals is made by two different tryptophan hydroxylases: TPH1 produces serotonin in the pineal gland and the enterochromaffin cells, while TPH2 produces it in the raphe nuclei and in the myenteric plexus. Genetically altered mice lacking TPH1 develop progressive loss of heart strength early on. They have pale skin and breathing difficulties, are easily tired, and eventually die of heart failure.][http://www.ncbi.nlm.nih.gov/pmc/articles/PMC263847/]
I actually wanted to make a whole list about topics like
cold exposure/omega-3/melatonin/cortisol/cooked protein/amphetamines/exercise/acidbase theory/sleep duration/gut flora/nuts etc. all things where I just have questions.
Studies that I read in the last 5 years, or experiences that I have, that just don't fit in the ray peat theory. But reading those studies again takes quite some time... Maybe another day.