And it appears they finally see the light. Maybe they've been reading the work of Ray Peat?
Scant Evidence Behind the Advice About Salt
Scant Evidence Behind the Advice About Salt
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Successful therapy of heart disease by high potassium together with low sodium in accord with predictions from the associated cation, structured wa... - PubMed - NCBIHigh potassium together with low sodium in diet and intravenous fluids has been observed clinically by Sodi-Pallares to have a beneficial effect on chronic heart failure and on acute myocardial infarction. Recent studies from the laboratory of Ling indicate that high potassium, low sodium environments can partially restore damaged cell proteins to their normal undamaged configuration. It follows that by this mechanism cell proteins damaged by the chronic or acute hypoxia of heart disease are probably partly repaired when high potassium, low sodium therapy is used.
They say there's scant evidence about salt restriction helping in heart failure.
Well,...
Successful therapy of heart disease by high potassium together with low sodium in accord with predictions from the associated cation, structured wa... - PubMed - NCBI
Or possibly low sodium with low potassium diets were harmful. What if the potassium intake was high?The evidence clearly shows that low sodium diets are harmful, especially for sick and elderly people.
Or possibly low sodium with low potassium diets were harmful. What if the potassium intake was high?
Yes, that's the worst situation, combining 2 problems.Or possibly low sodium with low potassium diets were harmful. What if the potassium intake was high?
I think Ray would disagree that Ling's in vitro experiments should be used to rationalize low-sodium diets in an in vivo context. The evidence clearly shows that low sodium diets are harmful, especially for sick and elderly people.
So, yes, I think the benefits of low sodium and high potassium diets would be even greater, if you made it a normal sodium high potassium diet.
Salt restriction also increases fasting norepinephrine, a substance that increases heart rate.
A review article by researchers at Boston University School of Medicine (BUSM) debunks the widely-believed concept that hypertension, or high blood pressure, is the result of excess salt causing an increased blood volume, exerting extra pressure on the arteries. Published online in the Journal of Hypertension, the study demonstrates that excess salt stimulates the sympathetic nervous system to produce adrenalin, causing artery constriction and hypertension.
The research was led by Irene Gavras, MD, and Haralambos Gavras, MD, both professors of medicine at BUSM.
"The purpose of this paper is to correct an erroneous concept that has prevailed for many years, even though scientific evidence has mounted against it," said Irene Gavras, who is also a physician in Boston Medical Center's Hypertension practice.
The term "volume-expanded hypertension" implies that excess salt leads to the retention of extra fluid within the arterial circulatory system, causing an increase in blood volume and added pressure on the arterial walls. However, research has shown that conditions characterized by the expansion of blood volume from other causes, such as the secretion of antidiuretic hormone or the excessive elevation of blood sugar, do not cause a rise in blood pressure because the extra fluid is accommodated by the distention of capillaries and veins.
"The body's circulatory system is a highly flexible vascular system with the capacity to open up new capillaries and distend veins in order to accommodate increased fluid volume," said Irene Gavras.
Through a review of numerous studies, the researchers demonstrated that the mechanism of hypertension resulting from the excessive consumption and retention of salt stimulates the sympathetic nervous system in the brain to increase adrenaline production. The increased adrenalin being circulated throughout the body causes the arteries to constrict, which results in resistance to blood flow and a decrease in circulatory volume.
The over-activation of the sympathetic nervous system - part of the autonomic nervous system that helps maintain the body's homeostasis - has been recognized clinically as a characteristic of hypertension that accompanies renal failure, which is the most typical example of elevated blood pressure from excessive salt retention. Diuretics, which remove excess salt, are widely used to treat this type of hypertension. However, this study provides convincing evidence that the sympathetic nervous system should be the focus of further investigations into treatments for hypertension.
"The implication of our findings shows that the optimal treatment for hypertension, for cases associated with renal failure, should not only include diuretics but also the use of drugs that block the central sympathetic nervous system," said Irene Gavras.
I think there was a thread here proving with scientific data that the mechanism by which salt increased blood pressure was by augmenting the secretion of adrenaline, causing a vasoconstriction, and not by an increased blood volume as everybody thought.
Salt Increases Adrenaline Study
Excess salt stimulates production of adrenalin and causes hypertension
Excess salt! What is excess salt?!I think there was a thread here proving with scientific data that the mechanism by which salt increased blood pressure was by augmenting the secretion of adrenaline, causing a vasoconstriction, and not by an increased blood volume as everybody thought.
Salt Increases Adrenaline Study
Excess salt stimulates production of adrenalin and causes hypertension
Heaney RP suggests an approach to set nutrient intake requirements that is based upon physiology.Excess salt! What is excess salt?!
Ray doesnt recommend excess salt! Thats my point.Heaney RP suggests an approach to set nutrient intake requirements that is based upon physiology.
"The requirement of a given nutrient can be said best when this intake calls for the least adaptation or compensation by a wealthy organism. For sodium, evidence indicates that such an intake is typically between 3000 and 5000 mg/d."
=> 3 gr Sodium = 7.5 gr salt. => Between 7.5 and 12 gr salt is optimal (if you get enough potassium too, 4.7 mg).
Source:
Heaney R.P. 2015. Making sense of the science of sodium. Nutr Today 50(2): 63-66.
Salt to tasteI'm not talking about excess but optimal range, what puts less stress onto hormones and organs.
Usually, yes. Most people can use "taste" as thermostat, when you're balanced.Salt to taste