Covid "Vaccine" Adverse Reaction Reports (Post Here)

[Beatrix_]

Earlier this week I went to the 2nd funeral this year due to turbo cancer, not counting the ones I read in the news killing celebs


View: https://twitter.com/P_McCulloughMD/status/1678848522517348356

 

[David PS]

View: https://twitter.com/P_McCulloughMD/status/1680213771829104644

 

[Jam]

View: https://twitter.com/ak_america/status/1681711657867943942

 

[Jam]

View: https://twitter.com/JohnBeaudoinSr/status/1682021371604262913

 

[Beatrix_]

View: https://twitter.com/mypillowgorl/status/1682172511197200384

 

[Beatrix_]

View: https://twitter.com/DowdEdward/status/1683252530594336769

 

[David PS]

Experts call for investigation as excess deaths spark ‘dangerous’ theories

The death toll in 2023 is nearing pandemic levels and the absence of an explanation is leading to wild theories. Experts warn that an urgent investigation into the matter is needed to curb dangerous theories and resulting anxieties.

www.express.co.uk

 

[Peatress]

Stop ‘gaslighting’ people left ill by Covid jabs, says BMJ study - Research Professional News

UK government must help people hurt by vaccines, say British Medical Journal study authors

www.researchprofessionalnews.com

 

[Athrunzala]

 

[Athrunzala]

 

[Athrunzala]

Bronny James in 'stable condition' after suffering cardiac arrest at basketball workout​

MSN

 

[Beatrix_]

View: https://twitter.com/stkirsch/status/1683945946609229824

 

[Beatrix_]

SARS-CoV-2 infection dysregulates NAD metabolism - PubMed

Our data suggest that infection perturbs NAD pathways, identify novel mechanisms that may explain some pathophysiology of CoVID-19 and suggest novel strategies for both treatment and prevention.

pubmed.ncbi.nlm.nih.gov

SARS-CoV-2 infection dysregulates NAD metabolism

Abstract
Introduction: Severe COVID-19 results initially in pulmonary infection and inflammation. Symptoms can persist beyond the period of acute infection, and patients with Post-Acute Sequelae of COVID (PASC) often exhibit a variety of symptoms weeks or months following acute phase resolution including continued pulmonary dysfunction, fatigue, and neurocognitive abnormalities. We hypothesized that dysregulated NAD metabolism contributes to these abnormalities.

Methods: RNAsequencing of lungs from transgenic mice expressing human ACE2 (K18-hACE2) challenged with SARS-CoV-2 revealed upregulation of NAD biosynthetic enzymes, including NAPRT1, NMNAT1, NAMPT, and IDO1 6 days post-infection.

Results: Our data also demonstrate increased gene expression of NAD consuming enzymes: PARP 9,10,14 and CD38. At the same time, SIRT1, a protein deacetylase (requiring NAD as a cofactor and involved in control of inflammation) is downregulated. We confirmed our findings by mining sequencing data from lungs of patients that died from SARS-CoV-2 infection. Our validated findings demonstrating increased NAD turnover in SARS-CoV-2 infection suggested that modulating NAD pathways may alter disease progression and may offer therapeutic benefits. Specifically, we hypothesized that treating K18-hACE2 mice with nicotinamide riboside (NR), a potent NAD precursor, may mitigate lethality and improve recovery from SARS-CoV-2 infection. We also tested the therapeutic potential of an anti- monomeric NAMPT antibody using the same infection model. Treatment with high dose anti-NAMPT antibody resulted in significantly decreased body weight compared to control, which was mitigated by combining HD anti-NAMPT antibody with NR. We observed a significant increase in lipid metabolites, including eicosadienoic acid, oleic acid, and palmitoyl carnitine in the low dose antibody + NR group. We also observed significantly increased nicotinamide related metabolites in NR treated animals.

Discussion: Our data suggest that infection perturbs NAD pathways, identify novel mechanisms that may explain some pathophysiology of CoVID-19 and suggest novel strategies for both treatment and prevention.

 

[charlie]

View: https://twitter.com/Inversionism/status/1686396871101583360

 

[Beatrix_]

View: https://twitter.com/EtanaHechtDC/status/1691053596161286144

 

[Beatrix_]

An unvaxxed family friend in his late 50s went to the hospital to investigate enlarged heart. He caught c-19 3x.

 

[Peatress]

@David PS posted this elsewhere

CDC Quietly Removes COVID Vaccine Adverse Events Reporting From Website

Even as many Americans suffer from COVID vaccine-related conditions, the CDC has removed the section from its website where such adverse reactions could be reported.

www.theepochtimes.com

When visiting the V-Safe page, a message shows: “Thank you for your participation. Data collection for COVID-19 vaccines concluded on June 30, 2023. If you have symptoms or health problems following your COVID-19 vaccination that concern you, please contact your healthcare provider. You can also report to the Vaccine Adverse Event Reporting System (VAERS).”

 

[David PS]

Covid-19 vaccination can induce multiple sclerosis via cross-reactive CD4+ T cells recognizing SARS-CoV-2 spike protein and myelin peptides | Multiple Sclerosis Journal; 28(3 Supplement):776, 2022. | EMBASE

pesquisa.bvsalud.org

 

[David PS]

Minds

www.minds.com

 

[Beatrix_]

SARS-CoV-2 reservoir in post-acute sequelae of COVID-19 (PASC) - Nature Immunology

Proal and colleagues review the evidence for long-term persistence of coronavirus SARS-CoV-2 in tissues of infected individuals and discuss how this viral reservoir may contribute to the pathogenesis of post-acute sequelae of COVID-19 (PASC).

www.nature.com

Review Article
Published: 04 September 2023
COVID-19

SARS-CoV-2 reservoir in post-acute sequelae of COVID-19 (PASC)

Nature Immunology (2023)

Abstract
Millions of people are suffering from Long COVID or post-acute sequelae of COVID-19 (PASC). Several biological factors have emerged as potential drivers of PASC pathology. Some individuals with PASC may not fully clear the coronavirus SARS-CoV-2 after acute infection. Instead, replicating virus and/or viral RNA—potentially capable of being translated to produce viral proteins—persist in tissue as a ‘reservoir’. This reservoir could modulate host immune responses or release viral proteins into the circulation. Here we review studies that have identified SARS-CoV-2 RNA/protein or immune responses indicative of a SARS-CoV-2 reservoir in PASC samples. Mechanisms by which a SARS-CoV-2 reservoir may contribute to PASC pathology, including coagulation, microbiome and neuroimmune abnormalities, are delineated. We identify research priorities to guide the further study of a SARS-CoV-2 reservoir in PASC, with the goal that clinical trials of antivirals or other therapeutics with potential to clear a SARS-CoV-2 reservoir are accelerated.