Westside PUFAs
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- Joined
- Feb 4, 2015
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because their early detection system (salivary amylase) isn't effective.
I don't think there is such a thing as an early detection system because there are no other cells other than whats on the tongue. Salt and sour cause the glands to salivate but that doesn't mean the pancreas starts pumping insulin. Even just being really hungry or thinking about food can cause salivation but that doesn't mean the pancreases detects sugar right in front of it. It has to pass by.
Their two quotes again:
"Because food is only in the mouth for a few seconds, oral amylolytic “predigestion” is often assumed to be of minimal importance, particularly given the presence of pancreatic amylase within the gastrointestinal tract.
"In contrast, pancreatic amylase, produced by the gene AMY2, has not undergone similar genetic repetition (15) even though the vast majority of starch digestion occurs in the small intestine via pancreatic amylase (16)"
Salivary amylase is produced by the salivary glands by default, regardless of what you eat. People produce varying amounts of it. But to claim that simply putting starch in your mouth is going to cause your pancreas to secret insulin is not true. The pancreas could already be in a pre-diabetic state in the first place, wasting insulin.
If they were just fat and insulin resistant, they shouldn't have performed similarly to the the HA group with the pure dextrose solution in both glucose tolerance and insulin release.
I don't regard a dextrose solution as I do a boiled potato with cofactors, or even white rice's own cofactor in its fluffy state, all the while consuming a low fat diet and not having excess adipose tissue. It's not a good study.