Amazoniac
Member
I might be in temporary overload of Tai Lopez and his Lamborghinis, what did you do?It just says <1 ppm
Here's what I did: 100mg of zinc gluconate/10^6 = 0.1mcg of cadmium
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I might be in temporary overload of Tai Lopez and his Lamborghinis, what did you do?It just says <1 ppm
I might be in temporary overload of Tai Lopez and his Lamborghinis, what did you do?
Here's what I did: 100mg of zinc gluconate/10^6 = 0.1mcg of cadmium
I have no idea if that's a high number, and if all that zinc is capable of impeding its absorption. Do you still supplement?I don't know lol. I guess that's not too good then...
I have no idea if that's a high number, and if all that zinc is capable of impeding its absorption. Do you still supplement?
It appears that Zn supplements with quite low Cd contamination ( <0.06 mcg Cd / 15 mg Zn ) can be produced."
Thanks!
Comparative absorption of zinc picolinate, zinc citrate and zinc gluconate in humans (Supported by GNC)Zinc gluconate is next to useless. Studies have shown its effect on zinc levels similar to placebo, and personally i found it to be the case.
5mg of zinc picolinate has a greater effect for me than 100mg of gluconate.
There was a small, insignificant rise in serum zinc during placebo supplementation
so what did you end up doing about the supplement Zinc?
oh ok. Thanks.zinc orotate ?
Previous work in this laboratory indicated that PA [picolinic acid] increases zinc uptake by both rat duodenal and ileal sacs incubated in vitro, but when it was incorporated into the diet of intact rats maintained in metabolic balance studies, it caused a marked increase in the urinary excretion of zinc (19). These observations suggest that PA increases the transport of zinc across membranes and may merely enhance the turnover of the metal without increasing its retention in the body.
In experiment 1 [oral] total zinc excretion was signficantly higher and zinc retention lower in experimental [picolinic acid] animals than control animals, so the experimental group was in a negative balance of approximately 8% of zinc intake during the collection period.
On each day of the collection period the PA-fed rats excreted a significantly greater proportion of (65)Zn than the corresponding control group (P < 0.001 for both experiments).
The fecal loss of orally administered (65)Zn was significantly greater from rats fed PA than the control group (P < 0.001) as shown in figure 2, but there was no significant difference in the specific activity of feces between the PA-fed and control groups of rats.
Effect of short-term feeding of PA to rats prelabeled with (65)Zn (expt 3 [a few weeks of accustomization to zinc supplementation followed by PA administration]). The rate of (65)Zn loss in the urine was greatly increased for 3 d when the rats were changed to the PA-containing diet (? < 0.001), but it returned to the same as the control animals after the control diet was reinstated on d 4 of the collection period (fig. 3). In both cases there was a delay of 24 h between the alteration in diet and the change in urinary excretion.
Effect of dietary PA on tissue levels of (65)Zn. The three experiments in this series all demonstrated that the effect of dietary PA was to increase the loss of (65)Zn from the body regardless of the route of administration of the isotope or the length of time of feeding the diet containing the ligand.
After absorption from the alimentary tract, PA is likely to interact most readily with zinc in the plasma, as indicated by the reduced activity of (65)Zn in the plasma of PA-fed rats in experiment 2 but not in 3 (table 4). Because of the equilibrium of zinc in plasma and cellular tissues, PA will then be able to influence cellular zinc in an indirect way, but if the ligand readily enters cells it may also do so directly. Our evidence for a general effect of PA on many tissues suggests that it is acting in an indirect way rather than interacting with zinc in specific target organs.
The conclusion that dietary PA enhances the general turnover of zinc has certain implications for its possible use as a therapeutic agent in conditions of zinc deficiency. It may help to ameliorate such conditions in the short term, by mobilizing zinc from deeper pools and making it available, but it seems unlikely to increase zinc retention by the body. Moreover, if given without an adequate intake of zinc, it could exacerbate the depletion over a longer period.
If you have zinc:copper ratio 7:1 or 8:1 only from foods, then you don't need supplement with zinc. With Ray Peat approach Beef liver = extremely high copper content and also if you use alot of gelatin-collagen/cocoa etc. which is also high in copper. Then you will have way too high copper to zinc ratio. But people here don't think that zinc/copper imbalance can be an issue. So whatever..
Gelatin.Members that are trying to induce a zinc deficiency at all costs with a "synergistic program" of plenty of white potatoes, chocolate, mushrooms, mangos, guavas, dried prunes and apricots, coffee, and daily bite-size pieces instead of weekly ruminant liver meals;
I wonder if there was something unusual about the gluconate (or the subjects?) in this study. They show decreased levels in erythrocytes and serum, but other studies find:
For example, oysters love to concentrate it.