Both vitamin E and aspirin have been associated in epidemiological studies with lower iron saturation, however I don't think either one is considered an iron chelator. This study says that aspirin is in fact an iron chelator.
http://www.ncbi.nlm.nih.gov/pubmed/3335633
"...Since MDA production in this system is known to be affected by iron-chelating agents (enhanced at low concentration, inhibited at higher concentration), the iron-chelating properties of ASA were investigated. Conductivity titration curves of Fe(OH)3 added to water or ASA suggested that the ASA was complexing with iron. The presence of an iron-ASA complex was established by high pressure liquid chromatographic analysis of the solution from this study. We conclude that aspirin enhances MDA production by hepatic microsomes and mitochondria via an aspirin-iron chelate and that this represents at least one mechanism by which aspirin may produce liver damage."
The concentration of aspirin resulting in maximum chelation of iron was 2.3mM. In human doses, 30mg/kg - 90mg/kg gives concentrations of 1mM - 3mM, so to achieve 2.3mM you'd need about 60mg/kg dose.
http://www.ncbi.nlm.nih.gov/pubmed/3335633
"...Since MDA production in this system is known to be affected by iron-chelating agents (enhanced at low concentration, inhibited at higher concentration), the iron-chelating properties of ASA were investigated. Conductivity titration curves of Fe(OH)3 added to water or ASA suggested that the ASA was complexing with iron. The presence of an iron-ASA complex was established by high pressure liquid chromatographic analysis of the solution from this study. We conclude that aspirin enhances MDA production by hepatic microsomes and mitochondria via an aspirin-iron chelate and that this represents at least one mechanism by which aspirin may produce liver damage."
The concentration of aspirin resulting in maximum chelation of iron was 2.3mM. In human doses, 30mg/kg - 90mg/kg gives concentrations of 1mM - 3mM, so to achieve 2.3mM you'd need about 60mg/kg dose.