Amazoniac
Member
@extremecheddar
If you don't mind sharing, what happened?
If you don't mind sharing, what happened?
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@extremecheddar
If you don't mind sharing, what happened?
Thank you for sharing. Have you tried everything that should've worked in combination?Still have discomfort. Eating lower fat , lower sugar, high starch is the only thing that gives relief. Taurine helps too. Seriously considering the
surgery.
My mother had the same issue at my age and the surgery fixed all her health problems. She was able to live a normal life and eat regular foods again.
Thanks for checking in.
Magnesium sulfate stimulates the gallbladder to contract. The grapefruit juice and the olive oil form a soap-like material. What you find in the toilet is "soap" colored green from the bile.Basically you fast for the morning and afternoon then take some Epsom salts in water at 6:00 p.m. , 8:00 p.m. then drink a mixture of 1/2 cup fresh grapefruit juice and 1/2 cup olive oil. Make sure you take L-ornithine from the Dr. Clark Store to put you to sleep and in the morning you take a couple more doses of Epsom salts/water.
According to Clark, the magnesium sulphate relaxes the bile ducts and opens them up so the gallstones and liver stones easily are pushed out. Expect to find parasites in the toilet the next morning too. I once found a flat worm about the size of my pinky nail. This stuff is reality.
I have a lot of experience in this area. While some of the stones can be formed in the intestines, there are indeed "stones" expelled from the liver/gallbladder during a liver flush.Magnesium sulfate stimulates the gallbladder to contract. The grapefruit juice and the olive oil form a soap-like material. What you find in the toilet is "soap" colored green from the bile.
Can you post the procedure you used?O, also, I had a period where my stools had become very tan/light brown ( indicating very low bile flow ). Then during a flush I expelled a very large amount of bile to the point where it hurt pretty badly. Very solid circumstantial evidence that "something" had been blocking the bile from flowing and was dislodged during the liver flush.
Magnesium sulfate stimulates the gallbladder to contract. The grapefruit juice and the olive oil form a soap-like material. What you find in the toilet is "soap" colored green from the bile.
This was on German TV. Fast-forward to 18:00. First they show gallstones, and next what was expelled after the Moritz protocol. They say that they have tested the stuff that was expelled after the "gallbladder flush": it mainly consisted of fatty acids; substances you typically find in gallstones were missing.I laugh every time I hear someone say that grapefruit juice and olive oil can somehow magically form soap overnight in the intestines.
Magnesium sulfate also causes the gallbladder to contract.The magnesium sulfate relaxes the bile ducts so that whatever is expelled from the gallbaldder can travel through the ducts without causing pain. It also acts as a laxative turning your bowels to liquid so that by the time the stones/gravel/congestion reaches the toilet, there is very little poop with it and therefore much easier to see the stones in the toilet.
The olive oil causes very strong contractions of the gallbladder (since no fat has been eaten in 24 hours) which causes stones/gravel/congestion to be released. The grapefruit juice (or orange or lemon juice) is just for taste to help the mixture go down.
Gallstones are usually accompanied by liver impairment
What's New About K2, By C.Masterjohn
"While all K vitamins are fat-soluble, they are not all equally soluble in fat. Those with longer tails are more fat-soluble than those with shorter tails; for tails of equal length, saturated tails are more fat-soluble than unsaturated tails. K vitamins that are more fat-soluble are carried deeper in the core of chylomicrons, while those that are less fat-soluble are carried more toward the edges. Let’s take the three forms most commonly found in supplements as examples: K1, MK-4, and MK-7. We would expect to find MK-7 in the center of the chylomicron, MK-4 closer to the edges, and K1 in between the two (Schurgers and Vermeer, 2002).
Chylomicrons move in and out of the bloodstream rapidly, with a half-life of 15-20 minutes (César, 2006). This means that once we eat a meal, 95% of the chylomicrons that enter our blood are fully cleared in the first hour. Very few tissues actually take up the whole chylomicron. Instead, most tissues use the enzyme lipoprotein lipase (LPL) to siphon off its nutrients bit by bit. While LPL is best known for feeding the heart, skeletal muscle, and adipose tissue, it also feeds other tissues such as the lungs, kidneys, mammary glands, and brain (Kersten, 2014). LPL spreads across the capillary beds that feed our great diversity of tissues, allowing widespread access to the fat-soluble nutrients we ingest in a meal. Presumably, these tissues all have greater access to the nutrients carried closer to the edges of the chylomicrons, such as MK-4.
As these many tissues feast on the chylomicrons, the chylomicrons get smaller and smaller until they become chylomicron remnants. A small handful of tissues donate apolipoprotein E (ApoE) to the chylomicron remnants, and then use the LDL receptor and other related receptors to bind to the ApoE and take up the whole remnant. This allows them to score everything left in the particle right down to its chewy center. In this sense, ApoE is like the bait on a fishing line, and the receptor is like the hook. While the liver is best known for fishing out chylomicron remnants in this manner, our bones and spleen do as well. Our bones primarily derive nutrients through the uptake of whole lipoprotein particles, and take up about a fifth as many chylomicron remnants as our liver (Shearer, 2008). Thus, we should expect bone and liver to primarily have access to nutrients carried in the center of chylomicrons, including K1, but especially the MKs with longer tails, such as MK-7."
"If we compare the results of the 2012 study to the earlier 2002 study, we can surmise that the dose of MK-4 in the 2012 study was low enough that the initial LPL feast in the first hour fully distributed it to a variety of tissues so that it was all gone by two hours, and that MK-7 circulated for such a long time because, like MK-9, it was redistributed in LDL particles. We should expect from this that MK-4 is good at nourishing most tissues, but not very good at nourishing liver or bone. By contrast, we should expect that MK-7 is good at nourishing the liver and even better at nourishing bone.
At the present time, there is no direct support for this, but there are hints that it may be the case. Sato (2012) cited a Japanese paper as finding that 1.5 milligrams of MK-4, but not 500 μg, improved the carboxylation of osteocalcin. Not even the abstract seems to be available in English, so it is difficult to evaluate the study. Later, Nakamura (2014) showed that only 600 μg of MK-4 is needed, but in this study the researchers simply gave the same people higher and higher doses each week and waited for osteocalcin carboxylation to improve. For all we know, their lowest dose, 300 μg, would have worked if they had given it longer than a week. In seeming contrast to MK-4, MK-7 improves osteocalcin carboxylation with as little as 100 μg (Knapen, 2012; Inaba, 2015).
Placing these studies side by side, they seem to suggest that improvements in osteocalcin carboxylation require much lower doses of MK-7 than of MK-4. However, the studies had different designs and were conducted in different populations that may have had different nutritional needs and different responses to vitamin K supplementation. In fact, Inaba (2015) fed MK-7 for four weeks while Nakamura (2014) only fed each dose of MK-4 for one week. This alone could explain the difference. To date, no one has compared the osteocalcin response to MK-4 and MK-7 head-to-head."