About a month ago, the last of the two teeth identified to have hidden periodontal pocket were removed.
When my dentists told me to get a new dental scan, involved x-ray and thermography, and a focused scan called digital ct scan, I complied. I was glad I did, as results from this scan showed that I had 2 hidden pockets of periodontitis. I knew early on that it's better to have teeth pulled than to save teeth that were going to just be recurrent sites of periodontal infection. I had three teeth removed already, even though these teeth could be saved if I were more concerned about bragging rights of having a full set of dentures.
The three teeth were removed as far back as nine years ago. Then (and continuing till the present), I had been dealing with a case of essential hypertension. It started as far back as 2005. Four years on, I was getting impatient in not being able to resolve my hypertension, which was getting worse. I'd been to naturopaths both in the US and in the Philippines, but never conventional doctors, and they could offer no answers. I stopped seeing them, and decided on my own to have the three teeth removed.
I thought I had checked off my dental issues as a cause of my hypertension. It's only recently that the new dental scanners became available, and it was thanks to new dental scanning equipment that two latent periodontal teeth were discovered. In between then and now, 9 years, I focused on chelation of lead, which I suspected was in my kidneys, and which I suspected was causing albumin to be excreted in my urine at higher than normal levels. Truth be told, chelation worked, but only to a certain extent. When I started using vitamin c and magnesium about a year ago, I was at 240/140. It's since improved to 180/120 but it hasn't budged any lower for a long time.
For a month now, I've stopped my magnesium and vitamin C protocol, to observe the effects of removing the periodontal issues. I didn't expect my bp to improve quickly, so it was no surprise that it stayed pretty steady. My heart rate did nudge up, from waking up to 54 bpm to around 59-62 bpm. My temperature stayed the same at 37. The best thing that came out of this so far: my urine has become less acidic. Previously, it would dip into 5.5 pH, but now it's not going down lower than 5.8 (This is big, as when I say 5.5, it could be lower as my urine test strip goes down only to 5.5).
But for a month, I had stayed puzzled as to why my urine became even more foamy. It wasn't sudsy foamy, but where it used to be composed of larger bubbles, it now became more smaller bubbles. I just had to figure out, or at least try to explain why this is happening.
After much thinking and trying out different word searches in Google, I can at least make an educated guess as to why: Oxidized albumin is being urinated as plaque is being broken down. Oxidized albumin is one of the many components of plaque.
Albumin is an antioxidant. It was being used to counter the oxidative damage from the body's attempt to counter the effects of periodontal bacteria. This would explain why my serum neutrophils would increase from 53% to 74% during the time I discovered my blood pressure deviating from my used-to-be normal of 120/80. There was a continual production of free radicals by the neutrophils to neutralize bacteria. And the spillover of free radicals were being taken care by antioxidants, among them being albumin.
I may have blamed a high urine ACR (albumin/creatinine) ratio, a marker of chronic kidney disease, to lead toxicity in my kidneys ( lead causes albumin to not be resorbed back into blood in the kidney tubules). Instead, what's really going on could be that the albumin being excreted are really oxidized albumin, arising out of its use as an antioxidant. The urine was already foamy from the discharge of oxidized albumin when the periodontal issue was ongoing, but when the issue was resolved, the urine stayed foamy and even more so. And I think it's because there was more oxidized albumin coming off the plaque that's disintegrating.
I'm speculating still at this point. Time will tell if I'm right on this.
In the meantime, I have turned my attention away from chelating lead, using magnesium and vitamin C, towards descaling and rejuvenating my arteries, with the use of, among other supplements, vitamin C and lysine. Vitamin C is to improve the collagen matrix use in repairing the endothelial linings, and lysine, to attract the plaque from the endothelial linings, enabling the plaque to slowly breakdown. I do hope that this is done slowly, though, as I don't want to see huge chunks of plaque coming off and blocking arteries and arterioles and cause damage. By the way, that is from the Linus Pauling therapy.
I'm hoping I'm right on this. If I'm right, this could explain:
- why I have hypertension
- why my serum albumin is low
- why my serum ferritin is high (well, not exactly, but the periodontal infection would be a case of chronic infection, and this would cause ferritin levels to be high, to keep iron away from bacteria)
- why i have an abnormally high serum neutrophil value
- why my erection capability is diminished (plaque on the endothelial lining would block NO's effects; in addition, nitric oxide is being used up to make free radicals to kill off bacteria by phagocytes)
- why my heart rate, at the high 60s and low 70s, cannot do into the mid-80s;
-why uric acid is high (its antioxidant ability is needed)
- why my hair is thinning (plaque is blocking flow of nutrients; albumin, a carrier of nutrients, is in short supply, being used to counter oxidation)
Why, just not taking care of my teeth well could cause all this? Wouldn't it be amazing that it all boils down to one thing?
Meanwhile, here are some links:
That oxidized albumin is part of plaque: Serum Oxidized Albumin and Cardiovascular Mortality in Normoalbuminemic Hemodialysis Patients: A Cohort Study
Oxidized human serum albumin as a possible correlating factor in atherosclerosis: Oxidized human serum albumin as a possible correlation factor for atherosclerosis in a rural Japanese population: the results of the Yakumo Study
The use of the albumin redox state as a marker for various diseases: https://www.researchgate.net/public...buminomics_Oxidized_Albumin_in_Human_Diseases
The association of severe periodontitis and microalbuminuria and chronic kidney disease (CKD): http://www.phmed.umu.se/digitalAssets/50/50453_thesis-shabnam-jun-21.pdf
When my dentists told me to get a new dental scan, involved x-ray and thermography, and a focused scan called digital ct scan, I complied. I was glad I did, as results from this scan showed that I had 2 hidden pockets of periodontitis. I knew early on that it's better to have teeth pulled than to save teeth that were going to just be recurrent sites of periodontal infection. I had three teeth removed already, even though these teeth could be saved if I were more concerned about bragging rights of having a full set of dentures.
The three teeth were removed as far back as nine years ago. Then (and continuing till the present), I had been dealing with a case of essential hypertension. It started as far back as 2005. Four years on, I was getting impatient in not being able to resolve my hypertension, which was getting worse. I'd been to naturopaths both in the US and in the Philippines, but never conventional doctors, and they could offer no answers. I stopped seeing them, and decided on my own to have the three teeth removed.
I thought I had checked off my dental issues as a cause of my hypertension. It's only recently that the new dental scanners became available, and it was thanks to new dental scanning equipment that two latent periodontal teeth were discovered. In between then and now, 9 years, I focused on chelation of lead, which I suspected was in my kidneys, and which I suspected was causing albumin to be excreted in my urine at higher than normal levels. Truth be told, chelation worked, but only to a certain extent. When I started using vitamin c and magnesium about a year ago, I was at 240/140. It's since improved to 180/120 but it hasn't budged any lower for a long time.
For a month now, I've stopped my magnesium and vitamin C protocol, to observe the effects of removing the periodontal issues. I didn't expect my bp to improve quickly, so it was no surprise that it stayed pretty steady. My heart rate did nudge up, from waking up to 54 bpm to around 59-62 bpm. My temperature stayed the same at 37. The best thing that came out of this so far: my urine has become less acidic. Previously, it would dip into 5.5 pH, but now it's not going down lower than 5.8 (This is big, as when I say 5.5, it could be lower as my urine test strip goes down only to 5.5).
But for a month, I had stayed puzzled as to why my urine became even more foamy. It wasn't sudsy foamy, but where it used to be composed of larger bubbles, it now became more smaller bubbles. I just had to figure out, or at least try to explain why this is happening.
After much thinking and trying out different word searches in Google, I can at least make an educated guess as to why: Oxidized albumin is being urinated as plaque is being broken down. Oxidized albumin is one of the many components of plaque.
Albumin is an antioxidant. It was being used to counter the oxidative damage from the body's attempt to counter the effects of periodontal bacteria. This would explain why my serum neutrophils would increase from 53% to 74% during the time I discovered my blood pressure deviating from my used-to-be normal of 120/80. There was a continual production of free radicals by the neutrophils to neutralize bacteria. And the spillover of free radicals were being taken care by antioxidants, among them being albumin.
I may have blamed a high urine ACR (albumin/creatinine) ratio, a marker of chronic kidney disease, to lead toxicity in my kidneys ( lead causes albumin to not be resorbed back into blood in the kidney tubules). Instead, what's really going on could be that the albumin being excreted are really oxidized albumin, arising out of its use as an antioxidant. The urine was already foamy from the discharge of oxidized albumin when the periodontal issue was ongoing, but when the issue was resolved, the urine stayed foamy and even more so. And I think it's because there was more oxidized albumin coming off the plaque that's disintegrating.
I'm speculating still at this point. Time will tell if I'm right on this.
In the meantime, I have turned my attention away from chelating lead, using magnesium and vitamin C, towards descaling and rejuvenating my arteries, with the use of, among other supplements, vitamin C and lysine. Vitamin C is to improve the collagen matrix use in repairing the endothelial linings, and lysine, to attract the plaque from the endothelial linings, enabling the plaque to slowly breakdown. I do hope that this is done slowly, though, as I don't want to see huge chunks of plaque coming off and blocking arteries and arterioles and cause damage. By the way, that is from the Linus Pauling therapy.
I'm hoping I'm right on this. If I'm right, this could explain:
- why I have hypertension
- why my serum albumin is low
- why my serum ferritin is high (well, not exactly, but the periodontal infection would be a case of chronic infection, and this would cause ferritin levels to be high, to keep iron away from bacteria)
- why i have an abnormally high serum neutrophil value
- why my erection capability is diminished (plaque on the endothelial lining would block NO's effects; in addition, nitric oxide is being used up to make free radicals to kill off bacteria by phagocytes)
- why my heart rate, at the high 60s and low 70s, cannot do into the mid-80s;
-why uric acid is high (its antioxidant ability is needed)
- why my hair is thinning (plaque is blocking flow of nutrients; albumin, a carrier of nutrients, is in short supply, being used to counter oxidation)
Why, just not taking care of my teeth well could cause all this? Wouldn't it be amazing that it all boils down to one thing?
Meanwhile, here are some links:
That oxidized albumin is part of plaque: Serum Oxidized Albumin and Cardiovascular Mortality in Normoalbuminemic Hemodialysis Patients: A Cohort Study
Oxidized human serum albumin as a possible correlating factor in atherosclerosis: Oxidized human serum albumin as a possible correlation factor for atherosclerosis in a rural Japanese population: the results of the Yakumo Study
The use of the albumin redox state as a marker for various diseases: https://www.researchgate.net/public...buminomics_Oxidized_Albumin_in_Human_Diseases
The association of severe periodontitis and microalbuminuria and chronic kidney disease (CKD): http://www.phmed.umu.se/digitalAssets/50/50453_thesis-shabnam-jun-21.pdf
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